Letter to the Editor Calcinosis cutis in epidermal necrolysis: role of caspofungin?

Hester Colboc (1) , Thomas Bettuzzi (2) , Marine Badrignans (3) , Dominique Bazin , Antoine Boury , Emmanuel Letavernier (4) , Vincent Frochot (4) , Ellie Tang (4) , Philippe Moguelet (5) , Nicolas Ortonne (6) , Nicolas Prost (7) , Saskia Ingen‐housz‐oro (2)

1 CHU Rothschild [AP-HP]
2 Service de dermatologie [Mondor]
3 Département de pathologie [Mondor]
4 Des Maladies Rénales Rares aux Maladies Fréquentes, Remodelage et Réparation
5 Service d’Anatomopathologie [CHU Tenon]
6 CHU Henri Mondor [Créteil]
7 Service d'anesthésie-réanimation SAMU94-SMUR94 [Mondor]

Toxic epidermal necrolysis (TEN) is a rare life-threatening condition, usually drug-induced, characterised by a diffuse epidermal and mucosal detachment. Skin histology reveals pan epidermal necrolysis. Mucocutaneous healing usually lasts 2 to 3 weeks.1 Calcinosis cutis is reported in various skin diseases, with dystrophic calcification following tissue damage being its most common cause.2 We describe four patients who presented TEN and atypical healing retardation due to calcinosis cutis.

Domains: Dermatology

Hester Colboc, Thomas Bettuzzi, Marine Badrignans, Dominique Bazin, Antoine Boury, et al.. Letter to the Editor Calcinosis cutis in epidermal necrolysis: role of caspofungin?. Journal of the European Academy of Dermatology and Venereology, 2021, ⟨10.1111/jdv.17808⟩. ⟨hal-03426502⟩